Sensory abnormalities and pain in Parkinson disease and its modulation by treatment of motor symptoms

Author:

Cury R.G.123,Galhardoni R.1,Fonoff E.T.145,Perez Lloret S.6,dos Santos Ghilardi M.G.5,Barbosa E.R.3,Teixeira M.J.12345,Ciampi de Andrade D.124

Affiliation:

1. Pain Center; Department of Neurology; University of São Paulo; São Paulo Brazil

2. Pain Center; Instituto do Câncer do Estado de São Paulo; São Paulo Brazil

3. Movement Disorders Group; Department of Neurology; University of São Paulo; São Paulo Brazil

4. Transcranial Magnetic Stimulation Laboratory; Psychiatry Institute; University of São Paulo; São Paulo Brazil

5. Neurosurgery Division; Department of Neurology; University of São Paulo; São Paulo Brazil

6. Laboratory of Clinical Pharmacology and Epidemiology; Catholic University; Buenos Aires Argentina

Funder

Pain Center of the Department of Neurology, University of São Paulo

Publisher

Wiley

Subject

Anesthesiology and Pain Medicine

Reference122 articles.

1. Dopamine receptor antagonists in the nucleus accumbens attenuate analgesia induced by ventral tegmental area substance P or morphine and by nucleus accumbens amphetamine;Altier;J Pharmacol Exp Ther,1998

2. The role of dopamine in the nucleus accumbens in analgesia;Altier;Life Sci,1999

3. Disturbances of learning processes in the basal ganglia in the pathogenesis of Parkinson's disease: A novel theory;Baev;Neurol Res,1995

4. Treatment of advanced Parkinson's disease by posterior GPi pallidotomy: 1-year results of a pilot study;Baron;Ann Neurol,1996

5. Levodopa and induced-pain response: A study of patients with parkinsonian and pain syndromes;Battista;Arch Intern Med,1973

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