Metabotropic glutamate 2,3 receptor stimulation desensitizes agonist activation of G‐protein signaling and alters transcription regulators in mesocorticolimbic brain regions-Reference-Cited by-同舟云学术

Metabotropic glutamate 2,3 receptor stimulation desensitizes agonist activation of G‐protein signaling and alters transcription regulators in mesocorticolimbic brain regions

Published:2020-10-23 Issue:4 Volume:75 Page:
ISSN:0887-4476
Container-title:Synapse
language:en
Short-container-title:Synapse

Author:

Burgos‐Aguilar Carolina¹^ORCID,Ferris Mark J.¹,Sexton Lacey L.¹,Sun Haiguo¹,Xiao Ruoyu¹,Chen Rong¹,Childers Steven R.¹,Howlett Allyn C.¹^ORCID

Affiliation:

1. Department of Physiology and Pharmacology Center for the Neurobiology of Addiction TreatmentOne Medical Center Blvd.Wake Forest School of Medicine Winston‐Salem NC USA

Funder

National Institute on Drug Abuse

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/syn.22190

Reference39 articles.

1. mGlu2 Receptor Agonism, but Not Positive Allosteric Modulation, Elicits Rapid Tolerance towards Their Primary Efficacy on Sleep Measures in Rats

2. Rats with Extended Access to Cocaine Exhibit Increased Stress Reactivity and Sensitivity to the Anxiolytic-Like Effects of the mGluR 2/3 Agonist LY379268 during Abstinence

3. Preferential Effects of the Metabotropic Glutamate 2/3 Receptor Agonist LY379268 on Conditioned Reinstatement versus Primary Reinforcement: Comparison between Cocaine and a Potent Conventional Reinforcer

4. Chronic ▵9-Tetrahydrocannabinol Treatment Produces a Time-Dependent Loss of Cannabinoid Receptors and Cannabinoid Receptor-Activated G Proteins in Rat Brain

5. Tolerance to the motor impairment, but not to the reversal of PCP-induced motor activities by oral administration of the mGlu2/3 receptor agonist, LY379268

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