Spinal astrocytes stimulated by tumor necrosis factor-α and/or interferon-γ attenuate connexin 43-gap junction via c-jun terminal kinase activity
Author:
Affiliation:
1. Department of Pharmacology; Graduate School of Biomedical and Health Sciences; Hiroshima University; Hiroshima; Japan
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience
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