TGF-β signaling through SMAD2/3 induces the quiescent microglial phenotype within the CNS environment-Reference-Cited by-同舟云学术

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TGF-β signaling through SMAD2/3 induces the quiescent microglial phenotype within the CNS environment

Published:2012-04-17 Issue:7 Volume:60 Page:1160-1171
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Abutbul Shai,Shapiro Jenny,Szaingurten-Solodkin Irit,Levy Nitzan,Carmy Yaron,Baron Rona,Jung Steffen,Monsonego Alon

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/glia.22343/fullpdf

Reference57 articles.

1. Infiltrating monocytes trigger EAE progression, but do not contribute to the resident microglia pool;Ajami;Nat Neurosci,2011

2. Microglia and microglia-like cell differentiated from DC inhibit CD4 T cell proliferation;Bai;PLoS One,2009

3. Functional expression of the fractalkine (CX3C) receptor and its regulation by lipopolysaccharide in rat microglia;Boddeke;Eur J Pharmacol,1999

4. Loss of TGF-β 1 leads to increased neuronal cell death and microgliosis in mouse brain;Brionne;Neuron,2003

5. CD11c/EYFP transgene illuminates a discrete network of dendritic cells within the embryonic, neonatal, adult, and injured mouse brain;Bulloch;J Comp Neurol,2008

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