Myotonic dystrophy expanded CUG repeats disturb the expression and phosphorylation of τ in PC12 cells-Reference-Cited by-同舟云学术

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Myotonic dystrophy expanded CUG repeats disturb the expression and phosphorylation of τ in PC12 cells

Published:2006 Issue:4 Volume:84 Page:841-851
ISSN:0360-4012
Container-title:Journal of Neuroscience Research
language:en
Short-container-title:J. Neurosci. Res.

Author:

Hernández-Hernández Oscar,Bermúdez-de-León Mario,Gómez Pablo,Velázquez-Bernardino Prisiliana,García-Sierra Francisco,Cisneros Bulmaro

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Reference53 articles.

1. Microtubule-binding domain of tau proteins.

2. The myotonic dystrophy expanded CUG repeat tract is necessary but not sufficient to disrupt C2C12 myoblast differentiation

3. Myotonic Dystrophy as a Brain Disorder

4. Molecular basis of myotonic dystrophy: Expansion of a trinucleotide (CTG) repeat at the 3′ end of a transcript encoding a protein kinase family member

5. PC12 cells express juvenile microtubule-associated proteins during nerve growth factor-induced neurite outgrowth.

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1. Protein Phosphorylation Alterations in Myotonic Dystrophy Type 1: A Systematic Review;International Journal of Molecular Sciences;2023-02-04

2. Myotonic Dystrophy: an RNA Toxic Gain of Function Tauopathy?;Advances in Experimental Medicine and Biology;2019

3. High-content screening identifies small molecules that remove nuclear foci, affect MBNL distribution and CELF1 protein levels via a PKC-independent pathway in myotonic dystrophy cell lines;Human Molecular Genetics;2013-10-30

4. mTOR-dependent proliferation defect in human ES-derived neural stem cells affected by Myotonic Dystrophy Type1;Journal of Cell Science;2013-01-01

5. An SRp75/hnRNPG complex interacting with hnRNPE2 regulates the 5′ splice site of tau exon 10, whose misregulation causes frontotemporal dementia;Gene;2011-10

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