Pharmacology of mitochondrial permeability transition pore inhibitors
Author:
Affiliation:
1. Laboratory of Experimental CardiologyCardiology Research Institute, Tomsk National Research Medical Center of the Russian Academy of Science Tomsk Russia
2. Department of PathologyUniversity of Kentucky College of Medicine Lexington Kentucky
Funder
Russian Science Foundation
Publisher
Wiley
Subject
Drug Discovery
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/ddr.21593
Reference182 articles.
1. Cardiomyocyte Apoptosis Induced by Gαq Signaling Is Mediated by Permeability Transition Pore Formation and Activation of the Mitochondrial Death Pathway
2. An uncoupling channel within the c-subunit ring of the F1FO ATP synthase is the mitochondrial permeability transition pore
3. Bongkrekic Acid—a Review of a Lesser-Known Mitochondrial Toxin
4. Specific inhibition of the mitochondrial permeability transition prevents lethal reperfusion injury
5. Effect of intravenous TRO40303 as an adjunct to primary percutaneous coronary intervention for acute ST-elevation myocardial infarction: MITOCARE study results
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