Inhibition of microRNA‐92a increases blood vessels and satellite cells in skeletal muscle but does not improve duchenne muscular dystrophy–related phenotype inmdxmice-Reference-Cited by-同舟云学术

Inhibition of microRNA‐92a increases blood vessels and satellite cells in skeletal muscle but does not improve duchenne muscular dystrophy–related phenotype inmdxmice

Published:2019-02-23 Issue:5 Volume:59 Page:594-602
ISSN:0148-639X
Container-title:Muscle & Nerve
language:en
Short-container-title:Muscle Nerve

Author:

Verma Mayank¹^ORCID,Asakura Yoko²,Asakura Atsushi³^ORCID

Affiliation:

1. Medical Scientist Training ProgramUniversity of Minnesota Medical School Minneapolis Minnesota USA

2. Stem Cell InstituteUniversity of Minnesota Medical School Minneapolis Minnesota USA

3. Department of NeurologyUniversity of Minnesota Medical School Minneapolis Minnesota, 55455 USA

Funder

Muscular Dystrophy Association

National Institute of Arthritis and Musculoskeletal and Skin Diseases

Publisher

Wiley

Subject

Physiology (medical),Cellular and Molecular Neuroscience,Neurology (clinical),Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/mus.26433

Reference45 articles.

1. The Molecular Basis of Muscular Dystrophy in the mdx Mouse: a Point Mutation

2. Recovery of induced mutations for X chromosome-linked muscular dystrophy in mice.

3. Short Telomeres and Stem Cell Exhaustion Model Duchenne Muscular Dystrophy in mdx/mTR Mice

4. Myofiber-specific TEAD1 overexpression drives satellite cell hyperplasia and counters pathological effects of dystrophin deficiency

5. Flt-1 haploinsufficiency ameliorates muscular dystrophy phenotype by developmentally increased vasculature in mdx mice

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