Isoliquiritigenin inhibits microglia‐mediated neuroinflammation in models of Parkinson's disease via <scp>JNK</scp> / <scp>AKT</scp> / <scp>NFκB</scp> signaling pathway-Reference-Cited by-同舟云学术

Isoliquiritigenin inhibits microglia‐mediated neuroinflammation in models of Parkinson's disease via JNK / AKT / NFκB signaling pathway

Published:2022-12-09 Issue:3 Volume:37 Page:848-859
ISSN:0951-418X
Container-title:Phytotherapy Research
language:en
Short-container-title:Phytotherapy Research

Author:

Bai Yuyan¹,Zhou Jin¹,Zhu Han¹,Tao Yanlin¹,Wang Lupeng¹,Yang Liu¹,Wu Hui¹,Huang Fei¹,Shi Hailian¹^ORCID,Wu Xiaojun¹^ORCID

Affiliation:

1. Shanghai Key Laboratory of Compound Chinese Medicines, The Ministry of Education (MOE) Key Laboratory for Standardization of Chinese Medicines, The State Administration of TCM (SATCM) Key Laboratory for New Resources and Quality Evaluation of Chinese Medicine, Institute of Chinese Materia Medica Shanghai University of Traditional Chinese Medicine Shanghai People's Republic of China

Publisher

Wiley

Subject

Pharmacology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ptr.7665

Reference40 articles.

1. Cofilin Mediates LPS-Induced Microglial Cell Activation and Associated Neurotoxicity Through Activation of NF-κB and JAK–STAT Pathway

2. Pulsatile Stimulation Determines Timing and Specificity of NF-κB-Dependent Transcription

3. Lipopolysaccharide-Induced Neuroinflammation as a Bridge to Understand Neurodegeneration

4. Amentoflavone protects dopaminergic neurons in MPTP-induced Parkinson's disease model mice through PI3K/Akt and ERK signaling pathways

5. Olfaction in Parkinson's disease and related disorders

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