Changing the concepts of immune-mediated glomerular diseases through proteomics
Author:
Affiliation:
1. Department of Medicine; University of Louisville School of Medicine; Louisville KY USA
2. Robley Rex VA Medical Center; Louisville KY USA
Funder
U.S. Department of Veterans Affairs
Publisher
Wiley
Subject
Clinical Biochemistry
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/prca.201400159/fullpdf
Reference48 articles.
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2. KDIGO clinical practice guideline for glomerulonephritis;Kidney Disease: Improving Global Outcomes (KDIGO) Glomerulonephritis Work Group;Kidney Int. Suppl.,2012
3. Membranous nephropathy: from models to man;Beck;J. Clin. Invest.,2014
4. Complement-induced glomerular epithelial cell injury: the role of the membrane attack complex in rat membranous nephropathy;Cybulsky;J. Clin. Invest.,1986
5. All four putative ligand-binding domains in megalin contain pathogenic epitopes capable of inducing passive Heymann nephritis;Yamazaki;J. Am. Soc. Nephrol.,1998
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