GBE attenuates arsenite‐induced hepatotoxicity by regulating E2F1‐autophagy‐E2F7a pathway and restoring lysosomal activity-Reference-Cited by-同舟云学术

GBE attenuates arsenite‐induced hepatotoxicity by regulating E2F1‐autophagy‐E2F7a pathway and restoring lysosomal activity

Published:2020-11-10 Issue:5 Volume:236 Page:4050-4065
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Chen Xiong¹,Wang Dapeng¹,Sun Baofei¹,Liu Chunyan¹,Zhu Kai¹,Zhang Aihua¹^ORCID

Affiliation:

1. The Key Laboratory of Environmental Pollution Monitoring and Disease Control, Ministry of Education Guizhou Medical University Guiyang Guizhou China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.30147

Reference56 articles.

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2. Rho-Kinase Inhibitor Y-27632 Attenuates Arsenic Trioxide Toxicity in H9c2 Cardiomyoblastoma Cells

3. The regulatory role of COX-2 in the interaction between Cr(VI)-induced endoplasmic reticulum stress and autophagy in DF-1 cells

4. From the Cover: Arsenic Induces Accumulation of α-Synuclein: Implications for Synucleinopathies and Neurodegeneration

5. E2F7, a novel E2F featuring DP-independent repression of a subset of E2F-regulated genes

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