Nitidine from Zanthoxylum rhetsa and its cytotoxic activities in vitro and in silico ADMET properties

Author:

Tuyen Tran Thi1,Quan Pham Minh12,Nghi Do Huu12,Bach Pham Cao1,Hiep Dang Hoang3,Duong Nguyen Thanh4ORCID,Linh Nguyen Nhat5,Vien Trinh Anh6,Minh Pham Thi Hong12,Van Nguyen Thi Hong12ORCID

Affiliation:

1. Institute of Natural Product Chemistry (INPC) Vietnam Academy of Science and Technology Hanoi Vietnam

2. Graduate University of Science and Technology (GUST) Vietnam Academy of Science and Technology Hanoi Vietnam

3. University of Science and Technology of Hanoi (USTH) Vietnam Academy of Science and Technology Hanoi Vietnam

4. Institute for Tropical Technology (ITT) Vietnam Academy of Science and Technology Hanoi Vietnam

5. Hanoi‐Amsterdam High School for the Gifted, Class 12 Chemistry 1 Hanoi Vietnam

6. Department of Examination and Quality Assurance of Education Hanoi Medical University‐Thanh Hoa Campus Thanh Hoa Vietnam

Abstract

AbstractNitidine, a potential medicinal natural benzophenanthridine alkaloid, has expressed various bioactivities such as antibacterial, antifungal, antiviral, anti‐inflammatory, analgesic, and notably cytotoxicity. In this study, nitidine was isolated from trunk of Zanthoxylum rhetsa (Rutaceae) and its structure was elucidated by spectral data (1D and 2D NMR; and MS). The cytotoxicity of nitidine was assessed in vitro against five cancer cell lines, and normal cell line Vero. As the results, nitidine exhibited potent inhibitory activity against KB, LU‐1, HepG2, LNCaP and MCF7 cell lines with IC50 values of 0.28, 0.26, 0.27, 0.25 and 0.28 µm, respectively, while low‐cytotoxicity against normal cell line Vero with IC50 value of 140.65 µm. Comparing cytotoxic activity of nitidine with docking analysis in previous study, nitidine formed hydrogen bonds with residues Asn101 and Ala317 of tubulin and had a docking score of −14.45 kcal/mol, all the data proved nitidine would be a potential candidate for inhibiting the function of tubulin at the active site regarding binding affinity, dock pose, and ADMET‐where ADMET stands for absorption, distribution, metabolism, excretion and toxicity‐properties analysis.

Publisher

Wiley

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