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Deletion of transcription factor AP‐2β from the developing murine trabecular meshwork region leads to progressive glaucomatous changes

  • Published:2021-11-25 Issue:2 Volume:100 Page:638-652
  • ISSN:0360-4012
  • Container-title:Journal of Neuroscience Research
  • language:en
  • Short-container-title:J of Neuroscience Research

Author:

Taiyab Aftab1,Akula Monica1,Dham Japnit1,Deschamps Paula1,Sheardown Heather2,Williams Trevor3,Borrás Teresa4,West‐Mays Judith A.1ORCID

Affiliation:

1. Department of Pathology and Molecular Medicine McMaster University Hamilton ON Canada

2. Department of Chemical Engineering McMaster University Hamilton ON Canada

3. Department of Craniofacial Biology University of Colorado Aurora CO USA

4. Department of Ophthalmology University of North Carolina School of Medicine Chapel Hill NC USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Reference55 articles.

1. Reduction of intraocular pressure in mouse eyes treated with latanoprost;Aihara M.;Investigative Ophthalmology & Visual Science,2002

2. Amacrine cells coupled to ganglion cells via gap junctions are highly vulnerable in glaucomatous mouse retinas

3. AP-2β is required for formation of the murine trabecular meshwork and Schlemm's canal

4. Latanoprost in the treatment of glaucoma;Alm A.;Clinical Ophthalmology,2014

5. A Naturally Fluorescent Mgp Transgenic Mouse for Angiogenesis and Glaucoma Longitudinal Studies

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