Loss of Cbl-PI3K Interaction Enhances Osteoclast Survival due to p21-Ras Mediated PI3K Activation Independent of Cbl-b-Reference-Cited by-同舟云学术

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Loss of Cbl-PI3K Interaction Enhances Osteoclast Survival due to p21-Ras Mediated PI3K Activation Independent of Cbl-b

Published:2014-05-07 Issue:7 Volume:115 Page:1277-1289
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J. Cell. Biochem.

Author:

Adapala Naga Suresh¹,Barbe Mary F.²,Tsygankov Alexander Y.³,Lorenzo Joseph A.⁴,Sanjay Archana¹

Affiliation:

1. Department of Orthopedic Surgery; University of Connecticut Health Center; Farmington Connecticut 06032

2. Department of Anatomy and Cell Biology; Temple University School of Medicine; Philadelphia Pennsylvania 19140

3. Department of Microbiology and Immunology; Temple University School of Medicine; Philadelphia Pennsylvania 19140

4. Department of Medicine; University of Connecticut Health Center; Farmington Connecticut 06032

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/jcb.24779/fullpdf

Reference43 articles.

1. The loss of Cbl-phosphatidylinositol 3-kinase interaction perturbs RANKL-mediated signaling, inhibiting bone resorption and promoting osteoclast survival;Adapala;J Biol Chem,2010

2. Cbl-phosphatidylinositol 3 kinase interaction differentially regulates macrophage colony-stimulating factor-mediated osteoclast survival and cytoskeletal reorganization;Adapala;Ann NY Acad Sci,2010

3. A positive regulatory role for Cbl family proteins in tumor necrosis factor-related activation-induced cytokine (TRANCE) and CD40L-mediated akt activation;Arron;J Biol Chem,2001

4. Negative regulation of lymphocyte activation and autoimmunity by the molecular adaptor Cbl-b;Bachmaier;Nature,2000

5. Pathway crosstalk between Ras/Raf and PI3K in promotion of M-CSF-induced MEK/ERK-mediated osteoclast survival;Bradley;J Cell Biochem,2008

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