IL‐13 modulates ∆Np63 levels causing altered expression of barrier‐ and inflammation‐related molecules in human keratinocytes: A possible explanation for chronicity of atopic dermatitis
Author:
Affiliation:
1. Department of Pathology, School of Medicine Sapporo Medical University Sapporo Japan
2. Department of Dermatology, School of Medicine Sapporo Medical University Sapporo Japan
Funder
Japan Society for the Promotion of Science
Akiyama Life Science Foundation
Publisher
Wiley
Subject
Immunology,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/iid3.427
Reference50 articles.
1. Japanese guidelines for atopic dermatitis 2017
2. Association of Atopic Dermatitis With Sleep Quality in Children
3. The skin as an immune organ: Tolerance versus effector responses and applications to food allergy and hypersensitivity reactions
4. Barrier dysfunction in the atopic march—how does atopic dermatitis lead to asthma in children?
5. Efficacy and safety of dupilumab for moderate‐to‐severe atopic dermatitis: A systematic review for the EAACI biologicals guidelines
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