Genetic contribution of retinoid-related genes to neural tube defects

Author:

Li Huili12,Zhang Jing1,Chen Shuyuan3,Wang Fang2,Zhang Ting2,Niswander Lee1ORCID

Affiliation:

1. Department of Pediatrics; University of Colorado Anschutz Medical Campus; Children's Hospital Colorado; Aurora Colorado

2. Beijing Municipal Key Laboratory of Child Development and Nutriomics; Capital Institute of Pediatrics; Beijing China

3. Department of Pediatrics; XiangYa Hospital of Central South University; Changsha China

Funder

National Institutes of Health

National Natural Science Foundation of China

Eunice Kennedy Shriver National Institute of Child Health and Human Development

Publisher

Wiley

Subject

Genetics(clinical),Genetics

Reference72 articles.

1. The retinoic acid-metabolizing enzyme, CYP26A1, is essential for normal hindbrain patterning, vertebral identity, and development of posterior structures;Abu-Abed;Genes Development,2001

2. Genetic studies of ANKRD6 as a molecular switch between Wnt signaling pathways in human neural tube defects;Allache;Birth Defects Research A: Clinical and Molecular Teratology,2015

3. Retinoic acid-induced spina bifida: Evidence for a pathogenetic mechanism;Alles;Development,1990

4. Gene expression regulation by retinoic acid;Balmer;Journal of Lipid Research,2002

5. Nuclear retinoid receptors and the transcription of retinoid-target genes;Bastien;Gene,2004

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