Neutral ceramidase activity inhibition is involved in palmitate-induced apoptosis in INS-1 cells-Reference-Cited by-同舟云学术

Neutral ceramidase activity inhibition is involved in palmitate-induced apoptosis in INS-1 cells

Published:2017 Issue:8 Volume:64 Page:767-776
ISSN:0918-8959
Container-title:Endocrine Journal
language:en
Short-container-title:Endocr J

Author:

Luo Fen¹,Feng Yamin¹,Ma Huimin¹,Liu Chao²,Chen Guofang²,Wei Xiao²,Mao Xiaodong²,Li Xingjia²,Xu Yijiao²,Tang Shan¹,Wen Honghua¹,Jin Junfei³,Zhu Qun¹

Affiliation:

1. Department of Endocrinology, The Second Affiliated Hospital of Nanjing Medical University, Nanjing 210011, China

2. Endocrine and Diabetes Center, Jiangsu Province Hospital on Integration of Chinese and Western Medicine, Nanjing University of Traditional Chinese Medicine, Branch of China Academy of Chinese Medical Science, Nanjing 210028, China

3. China-USA Lipids in Health and Disease Research Center, Guilin Medical University, Gulin 541001, China

Publisher

Japan Endocrine Society

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism

Link

https://www.jstage.jst.go.jp/article/endocrj/64/8/64_EJ16-0512/_pdf

Reference27 articles.

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2. 2. Subramanian S, Chait A (2012) Hypertriglyceridemia secondary to obesity and diabetes. Biochim Biophys Acta 1821: 819-825.

3. 3. Zhou Y, Sun P, Wang T, Chen K, Zhu W, et al. (2015) Inhibition of Calcium Influx Reduces Dysfunction and Apoptosis in Lipotoxic Pancreatic beta-Cells via Regulation of Endoplasmic Reticulum Stress. PLoS One 10: e132411.

4. 4. Pardo V, Gonzalez-Rodriguez A, Muntane J, Kozma SC, Valverde AM (2015) Role of hepatocyte S6K1 in palmitic acid-induced endoplasmic reticulum stress, lipotoxicity, insulin resistance and in oleic acid-induced protection. Food Chem Toxicol 80: 298-309.

5. 5. Yoon JH, Kim D, Jang JH, Ghim J, Park S, et al. (2015) Proteomic analysis of the palmitate-induced myotube secretome reveals involvement of the annexin A1-formyl peptide receptor 2 (FPR2) pathway in insulin resistance. Mol Cell Proteomics 14: 882-892.

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