Hypercholesterolaemia induces early renal lesions characterized by upregulation of MMP-9 and iNOS and ET<SUB>A</SUB>R: alleviated by a dual endothelin receptor antagonist CPU0213 and simvastatin-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

Hypercholesterolaemia induces early renal lesions characterized by upregulation of MMP-9 and iNOS and ET_AR: alleviated by a dual endothelin receptor antagonist CPU0213 and simvastatin

Published:2009-06-01 Issue:6 Volume:61 Page:775-780
ISSN:0022-3573
Container-title:Journal of Pharmacy and Pharmacology
language:en
Short-container-title:j pharm pharmacol

Author:

Luo Lu,Dai De-Zai,Zheng Yu-Fen,Dai Yin

Publisher

Oxford University Press (OUP)

Subject

Pharmaceutical Science,Pharmacology

Cited by 10 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Histological, ultrastructural and immunohistochemical studies on the ameliorative role of Cinnamon zeylanicum against high cholesterol diet-induced hypercholesterolemia in the kidney of adult male albino rats;Heliyon;2022-08

2. Inhibition of CPU0213, a Dual Endothelin Receptor Antagonist, on Apoptosis via Nox4-Dependent ROS in HK-2 Cells;Cellular Physiology and Biochemistry;2016

3. Simvastatin Restored Vascular Reactivity, Endothelial Function and Reduced String Vessel Pathology in a Mouse Model of Cerebrovascular Disease;Journal of Cerebral Blood Flow & Metabolism;2015-03

4. AQP4 KO exacerbating renal dysfunction is mediated by endoplasmic reticulum stress and p66Shc and is attenuated by apocynin and endothelin antagonist CPU0213;European Journal of Pharmacology;2013-12

5. Free cholesterol accumulation impairs antioxidant activities and aggravates apoptotic cell death in menadione-induced oxidative injury;Archives of Biochemistry and Biophysics;2011-10

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP