IGF-1 secretion from PDPN-expressing stromal cells by CLEC-2 stimulation positively regulates erythropoiesis
Author:
Affiliation:
1. 山梨大学医学部臨床検査医学講座
Publisher
Japanese Society on Thrombosis and Hemostasis
Subject
General Medicine
Link
https://www.jstage.jst.go.jp/article/jjsth/33/4/33_2022_JJTH_33_4_466-473/_pdf
Reference10 articles.
1. 1) Suzuki-Inoue K, Fuller GLJ, García Á, et al.: Anovel Syk-dependent mechanism of platelet activation by the C-type lectin receptor CLEC-2. Blood 107: 542–549, 2006.
2. 2) Osada M, Inoue O, Ding G, et al.: Platelet activation receptor CLEC-2 regulates blood/lymphatic vessel separation by inhibiting proliferation, migration, and tube formation of lymphatic endothelial cells. J Biol Chem 287: 22241–22252, 2012.
3. 3) Shirai T, Inoue O, Tamura S, et al.: C-type lectin-like receptor 2 promotes hematogenous tumor metastasis and prothrombotic state in tumor-bearing mice. J Thromb Haemost 15: 513–525, 2017.
4. 4) Sasaki T, Shirai T, Tsukiji N, et al.: Functional characterization of recombinant snake venom rhodocytin: Rhodocytin mutant blocks CLEC-2/podoplanin-dependent platelet aggregation and lung metastasis. J Thromb Haemost 16: 960–972, 2018.
5. 5) Tsukiji N, Inoue O, Morimoto M, et al.: Platelets play an essential role in murine lung development through Clec-2/podoplanin interaction. Blood 132: 1167–1179, 2018.
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