Targeting key angiogenic pathways with a bispecific Cross MA b optimized for neovascular eye diseases-Reference-Cited by-同舟云学术

Targeting key angiogenic pathways with a bispecific Cross MA b optimized for neovascular eye diseases

Published:2016-10-14 Issue:11 Volume:8 Page:1265-1288
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Regula Jörg T¹,Lundh von Leithner Peter²,Foxton Richard²³,Barathi Veluchamy A⁴⁵,Cheung Chui Ming Gemmy⁴,Bo Tun Sai Bo⁴,Wey Yeo Sia⁴,Iwata Daiju²,Dostalek Miroslav³,Moelleken Jörg¹,Stubenrauch Kay G¹,Nogoceke Everson³,Widmer Gabriella³,Strassburger Pamela³,Koss Michael J⁶⁷,Klein Christian⁸,Shima David T²,Hartmann Guido³^ORCID

Affiliation:

1. Roche Pharma Research and Early Development Roche Innovation Center München Penzberg Germany

2. Department of Ocular Biology and Therapeutics UCL London Institute of Ophthalmology London UK

3. Roche Pharma Research and Early Development Roche Innovation Center Basel F. Hoffmann‐La Roche Ltd Basel Switzerland

4. Translational Pre‐Clinical Model Platform Singapore Eye Research Institute The Academia Singapore Singapore

5. The Ophthalmology & Visual Sciences Academic Clinical Program DUKE‐NUS Graduate Medical School Singapore Singapore

6. Department of Ophthalmology Goethe University Frankfurt am Main Germany

7. Department of Ophthalmology Ruprecht Karls University Heidelberg Germany

8. Roche Pharma Research and Early Development Roche Innovation Center Zürich F. Hoffmann‐La Roche Ltd Zürich Switzerland

Funder

Roche

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201505889

Reference60 articles.

1. Chapter 6 Ocular Models of Angiogenesis

2. Tie2 Receptor Ligands, Angiopoietin-1 and Angiopoietin-2, Modulate VEGF-Induced Postnatal Neovascularization

3. Control of vascular morphogenesis and homeostasis through the angiopoietin–Tie system

4. The development of the rat model of retinopathy of prematurity

5. Crystal structures of the Tie2 receptor ectodomain and the angiopoietin-2–Tie2 complex

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