Activation of the epithelial sodium channel ( EN aC) leads to cytokine profile shift to pro‐inflammatory in labor-Reference-Cited by-同舟云学术

Activation of the epithelial sodium channel ( EN aC) leads to cytokine profile shift to pro‐inflammatory in labor

Published:2018-08-28 Issue:10 Volume:10 Page:
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Sun Xiao¹,Guo Jing Hui¹²³,Zhang Dan⁴,Chen Jun‐jiang¹²³,Lin Wei Yin¹,Huang Yun⁴,Chen Hui¹,Huang Wen Qing¹,Liu Yifeng⁴,Tsang Lai Ling¹,Yu Mei Kuen¹²,Chung Yiu Wa¹,Jiang Xiaohua¹,Huang Hefeng⁴⁵,Chan Hsiao Chang¹^ORCID,Ruan Ye Chun²^ORCID

Affiliation:

1. Epithelial Cell Biology Research Centre School of Biomedical Sciences Faculty of Medicine The Chinese University of Hong Kong Hong Kong China

2. Department of Biomedical Engineering Faculty of Engineering The Hong Kong Polytechnic University Hong Kong China

3. Department of Physiology School of Medicine Jinan University Guangzhou China

4. Department of Reproductive Endocrinology Women's Hospital School of Medicine Zhejiang University Hangzhou China

5. International Peace Maternal and Child Health Hospital Shanghai Jiao Tong University Shanghai China

Funder

National Natural Science Foundation of China

Hong Kong Polytechnic University

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201808868

Reference55 articles.

1. Influence of infection during pregnancy on fetal development

2. High-Content siRNA Screen Reveals Global ENaC Regulators and Potential Cystic Fibrosis Therapy Targets

3. BaldwinDA(2015)Gene Expression OmnibusGSE73685(https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE73685).

4. Onset of human preterm and term birth is related to unique inflammatory transcriptome profiles at the maternal fetal interface

5. Combinatory approaches prevent preterm birth profoundly exacerbated by gene-environment interactions

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