Caveolin‐1 deficiency induces a MEK ‐ ERK 1/2‐Snail‐1‐dependent epithelial–mesenchymal transition and fibrosis during peritoneal dialysis-Reference-Cited by-同舟云学术

Caveolin‐1 deficiency induces a MEK ‐ ERK 1/2‐Snail‐1‐dependent epithelial–mesenchymal transition and fibrosis during peritoneal dialysis

Published:2014-12-30 Issue:1 Volume:7 Page:102-123
ISSN:1757-4676
Container-title:EMBO Molecular Medicine
language:en
Short-container-title:EMBO Mol Med

Author:

Strippoli Raffaele¹²,Loureiro Jesús³,Moreno Vanessa¹,Benedicto Ignacio³,Pérez Lozano María Luisa³,Barreiro Olga¹,Pellinen Teijo¹,Minguet Susana¹,Foronda Miguel¹,Osteso Maria Teresa¹,Calvo Enrique⁴,Vázquez Jesús⁴,López Cabrera Manuel³,Pozo Miguel Angel¹

Affiliation:

1. Department of Vascular biology and Inflammation Centro Nacional de Investigaciones Cardiovasculares Madrid Spain

2. Department of Cellular Biotechnologies and Haematology Sapienza University of Rome Rome Italy

3. Molecular Biology Unit Hospital Universitario de la Princesa Instituto de Investigación Sanitaria Princesa (IP) Madrid Spain

4. CIBERehd Instituto de Salud Carlos III Madrid Spain

Publisher

EMBO

Subject

Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/emmm.201404127

Reference81 articles.

1. Caveolae regulate the nanoscale organization of the plasma membrane to remotely control Ras signaling

2. Epithelial to Mesenchymal Transition and Peritoneal Membrane Failure in Peritoneal Dialysis Patients: Pathologic Significance and Potential Therapeutic Interventions

3. Mesenchymal Conversion of Mesothelial Cells as a Mechanism Responsible for High Solute Transport Rate in Peritoneal Dialysis: Role of Vascular Endothelial Growth Factor

4. Characterization of Epithelial-to-Mesenchymal Transition of Mesothelial Cells in a Mouse Model of Chronic Peritoneal Exposure to High Glucose Dialysate

5. Functionally specialized junctions between endothelial cells of lymphatic vessels

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