The alanine‐serine‐cysteine‐1 (Asc‐1) transporter controls glycine levels in the brain and is required for glycinergic inhibitory transmission-Reference-Cited by-同舟云学术

The alanine‐serine‐cysteine‐1 (Asc‐1) transporter controls glycine levels in the brain and is required for glycinergic inhibitory transmission

Published:2015-03-09 Issue:5 Volume:16 Page:590-598
ISSN:1469-221X
Container-title:EMBO reports
language:en
Short-container-title:EMBO Rep

Author:

Safory Hazem¹,Neame Samah¹,Shulman Yoav¹,Zubedat Salman¹,Radzishevsky Inna¹,Rosenberg Dina¹,Sason Hagit¹,Engelender Simone¹,Avital Avi¹²,Hülsmann Swen³,Schiller Jackie¹,Wolosker Herman¹

Affiliation:

1. The Rappaport Faculty of Medicine and Research Institute Technion–Israel Institute of Technology Haifa Israel

2. Emek Medical Center Afula Israel

3. Department of Anesthesiology Emergency and Intensive Care Medicine and Center for Nanoscale Microscopy and Molecular Physiology of the Brain Georg‐August‐University Göttingen Germany

Publisher

EMBO

Subject

Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.15252/embr.201439561

Reference33 articles.

1. Glycine receptor heterogeneity in rat spinal cord during postnatal development.

2. Deletion of the Mouse Glycine Transporter 2 Results in a Hyperekplexia Phenotype and Postnatal Lethality

3. Mutations in the α1 subunit of the inhibitory glycine receptor cause the dominant neurologic disorder, hyperekplexia

4. Hyperekplexia associated with compound heterozygote mutations in the beta-subunit of the human inhibitory glycine receptor (GLRB)

5. Mutations in the gene encoding GlyT2 (SLC6A5) define a presynaptic component of human startle disease

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