Genetic Modifiers of Fetal Haemoglobin (HbF) and Phenotypic Severity in β-Thalassemia Patients-Reference-Cited by-同舟云学术

Genetic Modifiers of Fetal Haemoglobin (HbF) and Phenotypic Severity in β-Thalassemia Patients

Published:2018-11-26 Issue:5 Volume:18 Page:295-305
ISSN:1566-5240
Container-title:Current Molecular Medicine
language:en
Short-container-title:CMM

Author:

Razak S.A.A.¹,Murad N.A.A.¹,Masra F.²,Chong D.L.S.²,Abdullah N.¹,Jalil N.³,Alauddin H.³,Sabudin R.Z.A.R.³,Ithnin A.³,Khai L.C.²,Aziz N.A.⁴,Muda Z.⁵,Ibrahim H.⁵,Latiff Z.A.²

Affiliation:

1. UKM Medical Molecular Biology Institute, Jalan Yaacob Latif, 56000 Cheras, Kuala Lumpur, Malaysia

2. Department of Paediatrics, Faculty of Medicine, University Kebangsaan Malaysia (UKM), Jalan Yaacob Latif, 56000 Cheras, Kuala Lumpur, Malaysia

3. Department of Pathology, Faculty of Medicine, University Kebangsaan Malaysia (UKM), Jalan Yaacob Latif, 56000 Cheras, Kuala Lumpur, Malaysia

4. dInstitute for Medical Research (IMR), Jalan Pahang, 50586 Kuala Lumpur, Malaysia

5. Institute of Paediatrics, Hospital Kuala Lumpur, Jalan Pahang, 50586 Kuala Lumpur, Malaysia

Publisher

Bentham Science Publishers Ltd.

Subject

Molecular Biology,Molecular Medicine,General Medicine,Biochemistry

Cited by 7 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. MicroRNA-92a-3p-mediated inhibition of BCL11A upregulates γ-globin expression and inhibits oxidative stress and apoptosis in erythroid precursor cells;Hematology;2022-09-30

2. A Machine Learning Model for Predicting Fetal Hemoglobin Levels in Sickle Cell Disease Patients;Proceedings of Sixth International Congress on Information and Communication Technology;2021-09-24

3. Ineffective Erythropoiesis in β-Thalassaemia: Key Steps and Therapeutic Options by Drugs;International Journal of Molecular Sciences;2021-07-05

4. Association of HMIP1 C-893A polymorphism and disease severity in patients with sickle cell anemia;Hematology, Transfusion and Cell Therapy;2021-07

5. Hyperhaemolysis in a pregnant woman with a homozygous β 0 ‐thalassemia mutation and two genetic modifiers;Molecular Genetics & Genomic Medicine;2021-05-07