Blockade of Angiotensin II Type 1 Receptor Improves the Arrhythmia Morbidity in Mice With Left Ventricular Hypertrophy-Reference-Cited by-同舟云学术

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Blockade of Angiotensin II Type 1 Receptor Improves the Arrhythmia Morbidity in Mice With Left Ventricular Hypertrophy

Published:2006 Issue:3 Volume:70 Page:335-341
ISSN:1346-9843
Container-title:Circulation Journal
language:en
Short-container-title:Circ J

Author:

Zhang Cuntai¹²,Yasuno Shinji³,Kuwahara Koichiro³,Zankov Dimitar P.⁴,Kobori Atsushi²,Makiyama Takeru²,Horie Minoru⁴

Affiliation:

1. Department of Cardiology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology

2. Department of Cardiovascular Medicine, Kyoto University Graduate School of Medicine

3. Department of Medicine and Clinical Science, Kyoto University Graduate School of Medicine

4. Department of Cardiovascular and Respiratory Medicine, Shiga University of Medical Science

Publisher

Japanese Circulation Society

Subject

Cardiology and Cardiovascular Medicine,General Medicine

Link

http://www.jstage.jst.go.jp/article/circj/70/3/70_3_335/_pdf

Reference35 articles.

1. Risk of ventricular arrhythmias in left ventricular hypertrophy: The Framingham Heart Study

2. Hypertension and sudden death

3. Effect of Left Ventricular Hypertrophy and Its Regression on Ventricular Electrophysiology and Vulnerability to Inducible Arrhythmia in the Feline Heart

4. Regression of Left Ventricular Hypertrophy With Captopril Restores Normal Ventricular Action Potential Duration, Dispersion of Refractoriness, and Vulnerability to Inducible Ventricular Fibrillation

5. Restoration of Normal Ventricular Electrophysiology in Renovascular Hypertensive Rabbits after Treatment with Losartan

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3. Melatonin inhibits angiotensin II–induced atrial fibrillation through preventing degradation of Ang II Type I Receptor–Associated Protein (ATRAP);Biochemical Pharmacology;2022-08

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