Association Between Telomere G-Tail Length and Coronary Artery Disease or Statin Treatment in Patients With Cardiovascular Risks ― A Cross-Sectional Study ―
Author:
Affiliation:
1. Department of Cardiology, Fukuoka University School of Medicine
2. Department of Cellular and Molecular Biology, Hiroshima University
3. Collaborative Laboratory of Liquid Biopsy, Hiroshima University
Publisher
Japanese Circulation Society
Subject
Marketing,Organizational Behavior and Human Resource Management,Strategy and Management,Drug Discovery,Pharmaceutical Science,Pharmacology
Link
https://www.jstage.jst.go.jp/article/circrep/5/8/5_CR-23-0038/_pdf
Reference52 articles.
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2. 2. Blackburn EH. Structure and function of telomeres. Nature 1991; 350: 569–573, doi:10.1038/350569a0.
3. 3. Levy MZ, Allsopp RC, Futcher AB, Greider CW, Harley CB. Telomere end-replication problem and cell aging. J Mol Biol 1992; 225: 951–960, doi:10.1016/0022-2836(92)90096-3.
4. 4. Nezu T, Hosomi N, Takahashi T, Anno K, Aoki S, Shimamoto A, et al. Telomere G-tail length is a promising biomarker related to white matter lesions and endothelial dysfunction in patients with cardiovascular risk: A cross-sectional study. EBioMedicine 2015; 2: 960–967, doi:10.1016/j.ebiom.2015.05.025.
5. 5. Anno K, Hayashi A, Takahashi T, Mitsui Y, Ide T, Tahara H. Telomerase activation induces elongation of the telomeric single-stranded overhang, but does not prevent chromosome aberrations in human vascular endothelial cells. Biochem Biophys Res Commun 2007; 353: 926–932, doi:10.1016/j.bbrc.2006.12.112.
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