F1FO-ATPase Activity and ATP Dependence of Mitochondrial Energization in Proximal Tubules after Hypoxia/Reoxygenation-Reference-Cited by-同舟云学术

F1FO-ATPase Activity and ATP Dependence of Mitochondrial Energization in Proximal Tubules after Hypoxia/Reoxygenation

Published:2005-04-20 Issue:6 Volume:16 Page:1742-1751
ISSN:1046-6673
Container-title:Journal of the American Society of Nephrology
language:en
Short-container-title:JASN

Author:

Feldkamp Thorsten,Kribben Andreas,Weinberg Joel M.

Publisher

American Society of Nephrology (ASN)

Subject

Nephrology,General Medicine

Reference35 articles.

1. Mitochondrial dysfunction during hypoxia/reoxygenation and its correction by anaerobic metabolism of citric acid cycle intermediates

2. Anaerobic and aerobic pathways for salvage of proximal tubules from hypoxia-induced mitochondrial injury

3. Energetic Determinants of Tyrosine Phosphorylation of Focal Adhesion Proteins during Hypoxia/Reoxygenation of Kidney Proximal Tubules

4. Preservation of complex I function during hypoxia-reoxygenation-induced mitochondrial injury in proximal tubules

5. Feldkamp T, Kribben A, Weinberg JM. Assessment of mitochondrial membrane potential in proximal tubules after hypoxia/reoxygenation. Am J Physiol Renal Physiology Epub ahead of print December 29, 2004

Cited by 19 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Sugarcane ash and sugarcane ash-derived silica nanoparticles alter cellular metabolism in human proximal tubular kidney cells;Environmental Pollution;2023-09

2. Inhibition of ACSF2 protects against renal ischemia/reperfusion injury via mediating mitophagy in proximal tubular cells;Free Radical Biology and Medicine;2023-03

3. Diverse Roles of Mitochondria in Renal Injury from Environmental Toxicants and Therapeutic Drugs;International Journal of Molecular Sciences;2021-04-17

4. Proximal Tubular Oxidative Metabolism in Acute Kidney Injury and the Transition to CKD;Kidney360;2020-12-22

5. Activation of BNIP3-mediated mitophagy protects against renal ischemia–reperfusion injury;Cell Death & Disease;2019-09