Impaired Protein Stability of 11β-Hydroxysteroid Dehydrogenase Type 2: A Novel Mechanism of Apparent Mineralocorticoid Excess-Reference-Cited by-同舟云学术

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Impaired Protein Stability of 11β-Hydroxysteroid Dehydrogenase Type 2: A Novel Mechanism of Apparent Mineralocorticoid Excess

Published:2007-02-21 Issue:4 Volume:18 Page:1262-1270
ISSN:1046-6673
Container-title:Journal of the American Society of Nephrology
language:en
Short-container-title:JASN

Author:

Atanasov Atanas G.,Ignatova Irena D.,Nashev Lyubomir G.,Dick Bernhard,Ferrari Paolo,Frey Felix J.,Odermatt Alex

Publisher

American Society of Nephrology (ASN)

Subject

Nephrology,General Medicine

Reference37 articles.

1. Evidence for an Unidentified Steroid in a Child with Apparent Mineralocorticoid Hypertension

2. Human hypertension caused by mutations in the kidney isozyme of 11β–hydroxysteroid dehydrogenase

3. Several homozygous mutations in the gene for 11 beta-hydroxysteroid dehydrogenase type 2 in patients with apparent mineralocorticoid excess

4. A mutation in the HSD11B2 gene in a family with apparent mineralocorticoid excess

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