mRNA Modification and Mechanisms of Kidney Fibrosis
Author:
Funder
Japan Society for the Promotion of Science
AMED-CREST
Publisher
Ovid Technologies (Wolters Kluwer Health)
Reference8 articles.
1. Inhibition of METTL3 attenuates renal injury and inflammation by alleviating TAB3 m6A modifications via IGF2BP2-dependent mechanisms;Wang;Sci Transl Med.,2022
2. METTL3-mediated N6-methyladenosine mRNA modification and cGAS-STING pathway activity in kidney fibrosis;Tsai;J Am Soc Nephrol.,2024
3. Mitochondrial damage causes inflammation via cGAS-STING signaling in acute kidney injury;Maekawa;Cell Rep.,2019
4. Mitochondrial damage and activation of the STING pathway lead to renal inflammation and fibrosis;Chung;Cell Metab.,2019
5. Role of the cGAS-STING pathway in systemic and organ-specific diseases;Skopelja-Gardner;Nat Rev Nephrol.,2022
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