A computational model of induced pluripotent stem‐cell derived cardiomyocytes incorporating experimental variability from multiple data sources-Reference-Cited by-同舟云学术

A computational model of induced pluripotent stem‐cell derived cardiomyocytes incorporating experimental variability from multiple data sources

Published:2019-07-27 Issue:17 Volume:597 Page:4533-4564
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:J Physiol

Author:

Kernik Divya C.¹,Morotti Stefano²^ORCID,Wu HaoDi³,Garg Priyanka³,Duff Henry J.⁴,Kurokawa Junko⁵^ORCID,Jalife José⁶⁷,Wu Joseph C.³,Grandi Eleonora²^ORCID,Clancy Colleen E.¹^ORCID

Affiliation:

1. Department of Physiology and Membrane Biology Department of Pharmacology, School of Medicine University of California, Davis Davis CA USA

2. Department of Pharmacology School of Medicine University of California Davis CA USA

3. Stanford Cardiovascular Institute Department of Medicine Division of Cardiovascular Medicine Stanford University School of Medicine Stanford CA USA

4. Libin Cardiovascular Institute of Alberta Faculty of Medicine University of Calgary Calgary AB Canada

5. Department of Bio‐Informational Pharmacology School of Pharmaceutical Sciences University of Shizuoka Shizuoka Japan

6. Department of Internal Medicine Center for Arrhythmia Research Cardiovascular Research Center University of Michigan Ann Arbor MI USA

7. Centro Nacional de Investigaciones Cardiovasculares (CNIC), and CIBERV Madrid Spain

Funder

American Heart Association

National Science Foundation

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP277724

Reference101 articles.

1. Sequential dissection of multiple ionic currents in single cardiac myocytes under action potential-clamp

2. Reconstruction of the action potential of ventricular myocardial fibres

3. Drug-induced arrhythmia: pharmacogenomic prescribing?

4. Isogenic human pluripotent stem cell pairs reveal the role of a KCNH2 mutation in long-QT syndrome

5. Calcium Fluxes Involved in Control of Cardiac Myocyte Contraction

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