Comment on: ‘Physiological and pathophysiological roles of the KCNK3 potassium channel in the pulmonary circulation and the heart’ by Saint‐Martin Willer et al.
Author:
Affiliation:
1. Department of Cardiology University Hospital Heidelberg Heidelberg Germany
2. DZHK (German Center for Cardiovascular Research), partner site Heidelberg/Mannheim University of Heidelberg Heidelberg Germany
Funder
Else Kröner-Fresenius-Stiftung
Publisher
Wiley
Link
https://physoc.onlinelibrary.wiley.com/doi/pdf/10.1113/JP286611
Reference6 articles.
1. Differential effects of volatile and intravenous anesthetics on the activity of human TASK-1
2. Physiological and pathophysiological roles of the KCNK3 potassium channel in the pulmonary circulation and the heart
3. A Non-canonical Voltage-Sensing Mechanism Controls Gating in K2P K+ Channels
4. Upregulation of K 2P 3.1 K + Current Causes Action Potential Shortening in Patients With Chronic Atrial Fibrillation
5. Inverse remodelling of K2P3.1 K+ channel expression and action potential duration in left ventricular dysfunction and atrial fibrillation: Implications for patient‐specific antiarrhythmic drug therapy;Schmidt C.;European Heart Journal,2017
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Correction to ‘Physiological and pathophysiological roles of the KCNK3 potassium channel in the pulmonary circulation and the heart’;The Journal of Physiology;2024-04-28
2. Response to a Letter to the Editor: Physiological and pathophysiological roles of the KCNK3 potassium channel in the pulmonary circulation and the heart;The Journal of Physiology;2024-04-22
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