Increased mitochondrial emission of reactive oxygen species and calpain activation are required for doxorubicin-induced cardiac and skeletal muscle myopathy
Author:
Affiliation:
1. Department of Applied Physiology & Kinesiology; University of Florida; Gainesville FL USA
2. Department of Pharmacology; Weill Cornell Medical College; New York NY USA
3. School of Kinesiology; Auburn University; Auburn AL USA
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1113/jphysiol.2014.286518/fullpdf
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3. Mitochondria-targeted plastoquinone derivatives as tools to interrupt execution of the aging pro-gram. 5. SkQ1 prolongs lifespan and prevents development of traits of senescence;Anisimov;Biochemistry (Mosc),2008
4. Adriamycin-induced oxidative mitochondrial cardiotoxicity;Berthiaume;Cell Biol Toxicol,2007
5. Targeting mitochondrial cardiolipin and the cytochrome c/cardiolipin complex to promote electron transport and optimize mitochondrial ATP synthesis;Birk;Br J Pharmacol,2014
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