Exposure to high fructose corn syrup during adolescence in the mouse alters hepatic metabolism and the microbiome in a sex‐specific manner-Reference-Cited by-同舟云学术

Exposure to high fructose corn syrup during adolescence in the mouse alters hepatic metabolism and the microbiome in a sex‐specific manner

Published:2021-02-04 Issue:5 Volume:599 Page:1487-1511
ISSN:0022-3751
Container-title:The Journal of Physiology
language:en
Short-container-title:J Physiol

Author:

Bhat Shazia F.¹,Pinney Sara E.²,Kennedy Katherine M.³^ORCID,McCourt Cole R.⁴,Mundy Miles A.⁵,Surette Michael G.³⁶⁷^ORCID,Sloboda Deborah M.³^ORCID,Simmons Rebecca A.⁶⁸^ORCID

Affiliation:

1. Department of Pediatrics Christiana Care Health System Newark DE USA

2. Division of Endocrinology and Diabetes Department of Pediatrics Children's Hospital of Philadelphia Philadelphia PA USA

3. Department of Biochemistry & Biomedical Sciences Farncombe Family Digestive Health Research Institute, McMaster University Hamilton Ontario Canada

4. School of Arts and Sciences University of Pennsylvania PA USA

5. Department of Pathobiology Brown University

6. Department of Medicine McMaster University Hamilton Ontario Canada

7. Farncombe Family Digestive Health Research Institute McMaster University Hamilton Ontario Canada

8. Department of Pediatrics Perelman School of Medicine, University of Pennsylvania, Children's Hospital of Philadelphia PA USA

Funder

National Institutes of Health

University of Pennsylvania

Publisher

Wiley

Subject

Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1113/JP280034

Reference98 articles.

1. Fructose-Induced Fatty Liver Disease

2. Deletion of Fructokinase in the Liver or in the Intestine Reveals Differential Effects on Sugar-Induced Metabolic Dysfunction

3. Thymosin alpha 1 attenuates lipid peroxidation and improves fructose-induced steatohepatitis in rats

4. Generation of Multimillion-Sequence 16S rRNA Gene Libraries from Complex Microbial Communities by Assembling Paired-End Illumina Reads

5. Fructose as a key player in the development of fatty liver disease

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