KDM5B Is Essential for the Hyperactivation of PI3K/AKT Signaling in Prostate Tumorigenesis-Reference-Cited by-同舟云学术

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KDM5B Is Essential for the Hyperactivation of PI3K/AKT Signaling in Prostate Tumorigenesis

Published:2020-08-31 Issue:21 Volume:80 Page:4633-4643
ISSN:0008-5472
Container-title:Cancer Research
language:en
Short-container-title:Cancer Res

Author:

Li Guoliang,Kanagasabai Thanigaivelan,Lu Wenfu,Zou Mike R.,Zhang Shang-Min,Celada Sherly I.,Izban Michael G.^ORCID,Liu Qi,Lu Tao,Ballard Billy R.^ORCID,Zhou Xinchun,Adunyah Samuel E.,Matusik Robert J.,Yan Qin^ORCID,Chen Zhenbang

Funder

NIH

Publisher

American Association for Cancer Research (AACR)

Subject

Cancer Research,Oncology

Cited by 39 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Challenges of targeting tumor microenvironment in prostate cancer;New Updates in Tumor Microenvironment [Working Title];2024-08-14

2. Epigenetic mechanism of RBM15 in affecting cisplatin resistance in laryngeal carcinoma cells by regulating ferroptosis;Biology Direct;2024-07-23

3. Discovery of an efficacious KDM5B PROTAC degrader GT-653 up-regulating IFN response genes in prostate cancer;European Journal of Medicinal Chemistry;2024-06

4. KDM5B promotes SMAD4 loss-driven drug resistance through activating DLG1/YAP to induce lipid accumulation in pancreatic ductal adenocarcinoma;Cell Death Discovery;2024-05-24

5. MicroRNAs as the critical regulators of epithelial mesenchymal transition in pancreatic tumor cells;Heliyon;2024-05

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