ATP-sensitive potassium currents from channels formed by Kir6 and a modified cardiac mitochondrial SUR2 variant
Author:
Publisher
Informa UK Limited
Subject
Biochemistry,Biophysics
Link
http://www.tandfonline.com/doi/pdf/10.4161/chan.26181
Reference40 articles.
1. ATP-regulated K+ channels in cardiac muscle
2. Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium.
3. Blockade of ATP-sensitive potassium channels prevents myocardial preconditioning in dogs.
4. Preconditioning the Myocardium: From Cellular Physiology to Clinical Cardiology
5. A Novel Sulfonylurea Receptor Forms with BIR (Kir6.2) a Smooth Muscle Type ATP-sensitive K+ Channel
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1. Cardiac overexpression of a mitochondrial SUR2A splice variant impairs cardiac function and worsens myocardial ischemia reperfusion injury in female mice;Journal of Molecular and Cellular Cardiology Plus;2024-09
2. Overexpression of a Short Sulfonylurea Splice Variant Increases Cardiac Glucose Uptake and Uncouples Mitochondria by Regulating ROMK Activity;Life;2023-04-14
3. The short form of the SUR1 and its functional implications in the damaged brain;Neural Regeneration Research;2022
4. Insulin down-regulates cardioprotective SUR2A in the heart-derived H9c2 cells: A possible explanation for some adverse effects of insulin therapy;Biochemistry and Biophysics Reports;2018-12
5. Transgenic overexpression of the SUR2A-55 splice variant in mouse heart reduces infract size and promotes protective mitochondrial function;Heliyon;2018-07
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