Chemotherapy-elicited upregulation of NKG2D and DNAM-1 ligands as a therapeutic target in multiple myeloma
Author:
Publisher
Informa UK Limited
Subject
Oncology,Immunology,Immunology and Allergy
Link
http://www.tandfonline.com/doi/pdf/10.4161/onci.26663
Reference10 articles.
1. NKG2D-Deficient Mice Are Defective in Tumor Surveillance in Models of Spontaneous Malignancy
2. Accelerated tumor growth in mice deficient in DNAM-1 receptor
3. The Requirement for DNAM-1, NKG2D, and NKp46 in the Natural Killer Cell-Mediated Killing of Myeloma Cells
4. ATM-ATR–dependent up-regulation of DNAM-1 and NKG2D ligands on multiple myeloma cells by therapeutic agents results in enhanced NK-cell susceptibility and is associated with a senescent phenotype
5. Senescence of Activated Stellate Cells Limits Liver Fibrosis
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