BET bromodomain-targeting compounds reactivate HIV from latency via a Tat-independent mechanism-Reference-Cited by-同舟云学术

BET bromodomain-targeting compounds reactivate HIV from latency via a Tat-independent mechanism

Published:2013-02 Issue:3 Volume:12 Page:452-462
ISSN:1538-4101
Container-title:Cell Cycle
language:en
Short-container-title:Cell Cycle

Author:

Boehm Daniela,Calvanese Vincenzo,Dar Roy D.,Xing Sifei,Schroeder Sebastian,Martins Laura,Aull Katherine,Li Pao-Chen,Planelles Vicente,Bradner James E.,Zhou Ming-Ming,Siliciano Robert F.,Weinberger Leor,Verdin Eric,Ott Melanie

Publisher

Informa UK Limited

Subject

Cell Biology,Developmental Biology,Molecular Biology

Link

http://www.tandfonline.com/doi/pdf/10.4161/cc.23309

Reference74 articles.

1. Latent infection of CD4+ T cells provides a mechanism for lifelong persistence of HIV-1, even in patients on effective combination therapy

2. Limits on Replenishment of the Resting CD4+ T Cell Reservoir for HIV in Patients on HAART

3. Hide-and-Seek: The Challenge of Viral Persistence in HIV-1 Infection

4. Prostratin Antagonizes HIV Latency by Activating NF-κB

5. Disulfiram Reactivates Latent HIV-1 in a Bcl-2-Transduced Primary CD4 + T Cell Model without Inducing Global T Cell Activation

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