Acetate metabolism does not reflect astrocytic activity, contributes directly to GABA synthesis, and is increased by silent information regulator 1 activation-Reference-Cited by-同舟云学术

Acetate metabolism does not reflect astrocytic activity, contributes directly to GABA synthesis, and is increased by silent information regulator 1 activation

Published:2017-01-23 Issue:6 Volume:140 Page:903-918
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Rowlands Benjamin D.¹²,Klugmann Matthias¹²^ORCID,Rae Caroline D.¹³

Affiliation:

1. Neuroscience Research Australia Randwick NSW Australia

2. Translational Neuroscience Facility School of Medical Sciences The University of New South Wales Sydney NSW Australia

3. School of Medical Sciences The University of New South Wales Sydney NSW Australia

Funder

Australian Research Council

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jnc.13916

Reference72 articles.

1. Nuclear-cytoplasmic localization of acetyl coenzyme a synthetase-1 in the rat brain

2. Role of SLC5A8, a plasma membrane transporter and a tumor suppressor, in the antitumor activity of dichloroacetate

3. Cerebral metabolism of acetate and glucose studied by 13C-n.m.r. spectroscopy. A technique for investigating metabolic compartmentation in the brain

4. Neuronal-glial metabolism under depolarizing conditions. A 13C-n.m.r. study

5. The turnover of glutamate, glutamine, aspartate and GABA labeled with [1-14C]acetate in caudate nucleus, thalamus and motor cortex (CAT)

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