Endothelin-1 contributes to the progression of renal injury in sickle cell disease via reactive oxygen species-Reference-Cited by-同舟云学术

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Endothelin-1 contributes to the progression of renal injury in sickle cell disease via reactive oxygen species

Published:2015-12-19 Issue:2 Volume:173 Page:386-395
ISSN:0007-1188
Container-title:British Journal of Pharmacology
language:en
Short-container-title:British Journal of Pharmacology

Author:

Brett Heimlich J¹,Speed Joshua S²,O'Connor Paul M¹,Pollock Jennifer S²,Townes Tim M³,Meiler Steffen E⁴,Kutlar Abdullah⁵,Pollock David M²

Affiliation:

1. Department of Physiology; Augusta University; Augusta GA 30912 USA

2. Cardio-Renal Physiology and Medicine, Department of Medicine; The University of Alabama at Birmingham; Birmingham AL 35233 USA

3. Department of Biochemistry; The University of Alabama at Birmingham; Birmingham AL 35233 USA

4. Department of Anesthesiology and Perioperative Medicine; Augusta University; Augusta GA 30912 USA

5. Department of Medicine, Medical College of Georgia; Augusta University; Augusta GA 30912 USA

Funder

National Heart, Lung, and Blood Institute

Publisher

Wiley

Subject

Pharmacology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/bph.13380/fullpdf

Reference68 articles.

1. Sickle cell nephropathy at end-stage renal disease in the United States: patient characteristics and survival;Abbott;Clin Nephrol,2002

2. The Concise Guide to PHARMACOLOGY 2013/14: G protein-coupled receptors;Alexander;Br J Pharmacol,2013

3. Sickle cell disease serum induces NADPH enzyme subunit expression and oxidant production in leukocytes;Almeida;Hematology,2010

4. Endothelium-restricted overexpression of human endothelin-1 causes vascular remodeling and endothelial dysfunction;Amiri;Circulation,2004

5. Heme triggers TLR4 signaling leading to endothelial cell activation and vaso-occlusion in murine sickle cell disease;Belcher;Blood,2014

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