Didox, a ribonucleotide reductase inhibitor, induces apoptosis and inhibits DNA repair in multiple myeloma cells
Author:
Publisher
Wiley
Subject
Hematology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-2141.2006.06261.x/fullpdf
Reference49 articles.
1. Ribonucleotide reductase M1 gene promoter activity, polymorphisms, population frequencies, and clinical relevance
2. A phase I and pharmacokinetic study of didox administered by 36 hour infusion
3. Constitutive Activation of Stat3 Signaling Confers Resistance to Apoptosis in Human U266 Myeloma Cells
4. Multiple myeloma cell adhesion-induced interleukin-6 expression in bone marrow stromal cells involves activation of NF-kappa B
5. Interleukin-6 Inhibits Fas-Induced Apoptosis and Stress-Activated Protein Kinase Activation in Multiple Myeloma Cells
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1. The Antitumor Didox Acts as an Iron Chelator in Hepatocellular Carcinoma Cells;Pharmaceuticals;2019-09-02
2. Didox (3,4-dihydroxybenzohydroxamic acid) suppresses IgE-mediated mast cell activation through attenuation of NFκB and AP-1 transcription;Cellular Immunology;2017-12
3. An in-silico approach to predict and exploit synthetic lethality in cancer metabolism;Nature Communications;2017-09-06
4. Didox (3,4-dihydroxybenzohydroxamic acid) suppresses IL-33-induced cytokine production in primary mouse mast cells;Cellular Immunology;2017-09
5. Clinical pharmacology and clinical trials of ribonucleotide reductase inhibitors: is it a viable cancer therapy?;Journal of Cancer Research and Clinical Oncology;2017-06-17
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