Inhibition of BK channels contributes to the second phase of the response to TRH in clonal rat anterior pituitary cells
Author:
Publisher
Wiley
Subject
Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-201X.2004.01266.x/fullpdf
Reference57 articles.
1. Overproduction of a Ca(2+)-independent protein kinase C isozyme, nPKC epsilon, increases the secretion of prolactin from thyrotropin-releasing hormone-stimulated rat pituitary GH4C1 cells.
2. Two Distinct Populations of Calcium Channels in a Clonal Line of Pituitary Cells
3. Effects of hypothalamic peptides on electrical activity and membrane currents of ‘patch perforated’ clamped GH3anterior pituitary cells
4. Characteristics and modulation by thyrotropin-releasing hormone of an inwardly rectifying K+ current in patch-perforated GH3 anterior pituitary cells
5. Protein phosphatase 2A reverses inhibiton of inward rectifying K+currents by thyrotropin-releasing hormone in GH3pituitary cells
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