M1 macrophages regulate TLR4/AP1 via paracrine to promote alveolar bone destruction in periodontitis-Reference-Cited by-同舟云学术

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M1 macrophages regulate TLR4/AP1 via paracrine to promote alveolar bone destruction in periodontitis

Published:2019-10-18 Issue:8 Volume:25 Page:1972-1982
ISSN:1354-523X
Container-title:Oral Diseases
language:en
Short-container-title:Oral Dis

Author:

Zhu Li‐Fang¹²³,Li Lu¹²,Wang Xiao‐Qian¹²,Pan Lu¹⁴,Mei You‐Min¹²,Fu Yong‐Wei¹²,Xu Yan¹²

Affiliation:

1. Jiangsu Key Laboratory of Oral Diseases Nanjing Medical University Nanjing China

2. Department of Periodontology The Affiliated Stomatological Hospital of Nanjing Medical University Nanjing China

3. Department of Stomatology The First Affiliated Hospital of Soochow University Suzhou China

4. Department of Basic Science of Stomatology The Affiliated Stomatological Hospital of Nanjing Medical University Nanjing China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

General Dentistry,Otorhinolaryngology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/odi.13167

Reference30 articles.

1. Regulation of the differentiation and function of osteoclasts

2. NF-κB addiction and its role in cancer: ‘one size does not fit all’

3. Human β-defensin 3 inhibits periodontitis development by suppressing inflammatory responses in macrophages

4. Periodontitis: a polymicrobial disruption of host homeostasis

5. Macrophage-elicited osteoclastogenesis in response toBrucella abortusinfection requires TLR2/MyD88-dependent TNF-α production

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