The hyperfibrinolytic state of mice with combined thrombin-activatable fibrinolysis inhibitor (TAFI) and plasminogen activator inhibitor-1 gene deficiency is critically dependent on TAFI deficiency-Reference-Cited by-同舟云学术

The hyperfibrinolytic state of mice with combined thrombin-activatable fibrinolysis inhibitor (TAFI) and plasminogen activator inhibitor-1 gene deficiency is critically dependent on TAFI deficiency

Published:2012-12 Issue:12 Volume:10 Page:2555-2562
ISSN:1538-7933
Container-title:Journal of Thrombosis and Haemostasis
language:en
Short-container-title:

Author:

VERCAUTEREN E.,PEETERS M.,HOYLAERTS M. F.,LIJNEN H. R.,MEIJERS J. C. M.,DECLERCK P. J.,GILS A.

Publisher

Wiley

Subject

Hematology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jth.12036/fullpdf

Reference43 articles.

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3. Identification of thrombin activatable fibrinolysis inhibitor (TAFI) in human platelets;Mosnier;Blood,2003

4. Plasminogen activator inhibitor type-1 - reactive center and amino-terminal heterogeneity determined by protein and cDNA sequencing;Andreasen;FEBS Lett,1986

5. Plasminogen activator inhibitor (PAI-1) in plasma and platelets;Booth;Br J Haematol,1988

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