Being in the red: a no-win situation with melanoma
Author:
Publisher
Wiley
Subject
Dermatology,General Biochemistry, Genetics and Molecular Biology,Oncology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/pcmr.12061/fullpdf
Reference5 articles.
1. Role of glutathione S-transferases in melanoma susceptibility: association with GSTP1 rs1695 polymorphism;Ibarrola-Villava;Br. J. Dermatol.,2012
2. The p16 (INK4A) tumor suppressor regulates cellular oxidative stress;Jenkins;Oncogene,2011
3. Alpha-melanocyte-stimulating hormone suppresses oxidative stress through a p53-mediated signaling pathway in human melanocytes;Kadekaro;Mol. Cancer Res.,2012
4. Melanoma induction by ultraviolet A but not ultraviolet B radiation requires melanin pigment;Noonan;Nat. Commun.,2012
5. UVA-induced oxidative degradation of melanins: fission of indole moiety in eumelanin and conversion to benzothiazole moiety in pheomelanin;Wakamatsu;Pigment Cell Melanoma Res.,2012
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2. Light-independent pro-inflammatory and pro-oxidant effects of purified human hair melanins on keratinocyte cell cultures;Experimental Dermatology;2017-01-09
3. Pheomelanin-induced oxidative stress: bright and dark chemistry bridging red hair phenotype and melanoma;Pigment Cell & Melanoma Research;2014-06-06
4. Red human hair pheomelanin is a potent pro-oxidant mediating UV-independent contributory mechanisms of melanomagenesis;Pigment Cell & Melanoma Research;2014-01-06
5. Simultaneous Purifying Selection on the Ancestral MC1R Allele and Positive Selection on the Melanoma-Risk Allele V60L in South Europeans;Molecular Biology and Evolution;2013-09-17
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