MyD88 signalling plays a critical role in host defence by controlling pathogen burden and promoting epithelial cell homeostasis during Citrobacter rodentium-induced colitis
Author:
Publisher
Hindawi Limited
Subject
Virology,Immunology,Microbiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1462-5822.2007.01071.x/fullpdf
Reference37 articles.
1. Myeloid differentiation factor 88-dependent signalling controls bacterial growth during colonization and systemic pneumococcal disease in mice
2. MyD88-deficient mice develop severe intestinal inflammation in dextran sodium sulfate colitis
3. Pretreatment of Mice with Streptomycin Provides a Salmonella enterica Serovar Typhimurium Colitis Model That Allows Analysis of Both Pathogen and Host
4. Myd88-dependent positioning of Ptgs2-expressing stromal cells maintains colonic epithelial proliferation during injury
5. Toll-Like Receptor 2 Controls Mucosal Inflammation by Regulating Epithelial Barrier Function
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