Mice genetically inactivated in interleukin-17A receptor are defective in long-term control ofMycobacterium tuberculosisinfection-Reference-Cited by-同舟云学术

Mice genetically inactivated in interleukin-17A receptor are defective in long-term control ofMycobacterium tuberculosisinfection

Published:2013-09-12 Issue:2 Volume:140 Page:220-231
ISSN:0019-2805
Container-title:Immunology
language:en
Short-container-title:Immunology

Author:

Freches Danielle¹,Korf Hannelie¹,Denis Olivier¹,Havaux Xavier²,Huygen Kris¹,Romano Marta¹

Affiliation:

1. Service Immunology; Scientific Institute of Public Health WIV-ISP (Site Ukkel); Brussels; Belgium

2. Cardiovascular Research Laboratory; Medical School; Université Catholique de Louvain; Brussels; Belgium

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/imm.12130/fullpdf

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2. Protective effect of BCG against tuberculous meningitis and miliary tuberculosis: a meta-analysis;Rodrigues;Int J Epidemiol,1993

3. How can immunology contribute to the control of tuberculosis ?;Kaufmann;Nat Rev Immunol,2001

4. An overview of IL-17 function and signaling;Gaffen;Cytokine,2008

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