Nitric oxide impacts on angiotensin AT 2 receptor modulation of high‐pressure baroreflex control of renal sympathetic nerve activity in anaesthetized rats
Author:
Affiliation:
1. Department of Physiology Western Gateway Building University College Cork Cork Ireland
Publisher
Wiley
Subject
Physiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/apha.12207
Reference47 articles.
1. Role of angiotensin AT2 receptors and nitric oxide in the cardiopulmonary baroreflex control of renal sympathetic nerve activity in rats;Abdulla M.H.;J Hypertens,2013
2. Role of brain angiotensin II in renal nerve inhibition elicited by volume expansion in the conscious rabbit
3. Lack of a centrally-mediated antihypertensive effect following acute or chronic central treatment with AT1-receptor antagonists in spontaneously hypertensive rats
4. Responses to GABA-A receptor blockade in the hypothalamic PVN are attenuated by local AT1 receptor antagonism
5. Nitric oxide modulation of glutamatergic, baroreflex, and cardiopulmonary transmission in the nucleus of the solitary tract
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