Effects of lesions and ganglioside GM1 treatment on striatal polyamine levels and nigral DA neurons. A role of putrescine in the neurotropic activity of gangliosides

Author:

AGNATI LUIGI F.,FUXE KJELL,ZINI ISABELLA,DAVALLI P.,CORTI A.,CALZA LAURA,TOFFANO GINO,ZOLI MICHELE,PICCININI GIORGIO,GOLDSTEIN MENEK

Publisher

Wiley

Subject

Physiology

Reference17 articles.

1. Gangliosides increase the survival of lesioned nigral dopamine neurons and favour the recovery of dopaminergic synaptic function in striatum of rats by collateral sprouting;Agnati;Acta Physiol Scand,1983

2. Chronic treatment with l-DOPA plus carbidopa in hemitransected rats: preferential effects at intact dopamine synapses leading to behavioural signs of dopamine receptor supersensitivity;Agnati;Acta Physiol Scand,1983

3. Agnati, L.F. , Fuxe, K. , Benfenati, F. , Toffano, G. , Cimino, M. , Battistini, N. , Calza, L. & Merlo Pich, E. 1984. Further studies on the effects of the GM1 ganglioside on the degenerative and regenerative features of mesostriatal dopamine neurons. Acta Physiol Scand Suppl. 532, 37-42.

4. Polyamine biosynthesis is required for survival of sympathetic neurons after axonal injury;Gilad;Brain Res,1983

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