SIRT1 but not its increased expression is essential for lifespan extension in caloric-restricted mice-Reference-Cited by-同舟云学术

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SIRT1 but not its increased expression is essential for lifespan extension in caloric-restricted mice

Published:2013-11-19 Issue:1 Volume:13 Page:193-196
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Mercken Evi M.¹,Hu Jia²,Krzysik-Walker Susan³,Wei Min²,Li Ying²,McBurney Michael W.⁴,de Cabo Rafael¹,Longo Valter D.²

Affiliation:

1. Translational Gerontology Branch; National Institute on Aging; National Institutes of Health; Baltimore MD 21224 USA

2. Davis School of Gerontology and Department of Biological Sciences; University of Southern California; Los Angeles CA 90089-2520 USA

3. Laboratory of Clinical Investigation; National Institute on Aging; National Institutes of Health; Baltimore MD 21224 USA

4. Department of Medicine; Ottawa Hospital Research Institute; University of Ottawa; Ottawa Ontario K1H 8L6 Canada

Publisher

Wiley

Subject

Cell Biology,Ageing

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/acel.12151/fullpdf

Reference14 articles.

1. Extending the lifespan of long-lived mice;Bartke;Nature,2001

2. Are sirtuins viable targets for improving healthspan and lifespan?;Baur;Nat. Rev.,2012

3. SirT1 regulates energy metabolism and response to caloric restriction in mice;Boily;PLoS One,2008

4. SIRT1 transgenic mice show phenotypes resembling calorie restriction;Bordone;Aging Cell,2007

5. Increase in activity during calorie restriction requires Sirt1;Chen;Science,2005

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