High molecular weight kininogen contributes to early mortality and kidney dysfunction in a mouse model of sickle cell disease-Reference-Cited by-同舟云学术

High molecular weight kininogen contributes to early mortality and kidney dysfunction in a mouse model of sickle cell disease

Published:2020-08-27 Issue:9 Volume:18 Page:2329-2340
ISSN:1538-7933
Container-title:Journal of Thrombosis and Haemostasis
language:en
Short-container-title:J Thromb Haemost

Author:

Sparkenbaugh Erica M.¹^ORCID,Kasztan Malgorzata²^ORCID,Henderson Michael W.¹,Ellsworth Patrick¹,Davis Parker Ross²,Wilson Kathryn J.¹,Reeves Brandi¹^ORCID,Key Nigel S.¹³^ORCID,Strickland Sidney⁴^ORCID,McCrae Keith⁵^ORCID,Pollock David M.²^ORCID,Pawlinski Rafal¹^ORCID

Affiliation:

1. UNC Blood Research Center Division of Hematology & Oncology Department of Medicine University of North Carolina at Chapel Hill Chapel Hill NC USA

2. Section of Cardio‐Renal Physiology and Medicine Division of Nephrology Department of Medicine University of Alabama at Birmingham Birmingham AL USA

3. Department of Pathology and Laboratory Medicine University of North Carolina at Chapel Hill Chapel Hill NC USA

4. Patricia and John Rosenwald Laboratory of Neurobiology and Genetics The Rockefeller University New York NY USA

5. Department of Hematology Oncology Taussig Cancer Institute Cleveland Clinic Cleveland OH USA

Funder

National Heart, Lung, and Blood Institute

American Heart Association

National Institute of Neurological Disorders and Stroke

Publisher

Wiley

Subject

Hematology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jth.14972

Reference51 articles.

1. Association of Coagulation Activation with Clinical Complications in Sickle Cell Disease

2. Hypercoagulability and thrombotic complications in hemolytic anemias

3. Tissue factor promotes activation of coagulation and inflammation in a mouse model of sickle cell disease

4. Heme induces endothelial tissue factor expression: potential role in hemostatic activation in patients with hemolytic anemia

5. Hypercoagulability in Sickle Cell Disease: New Approaches to an Old Problem

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