Status of treatment strategies for Hutchinson–Gilford progeria syndrome with a focus on prelamin: A posttranslational modification-Reference-Cited by-同舟云学术

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Status of treatment strategies for Hutchinson–Gilford progeria syndrome with a focus on prelamin: A posttranslational modification

Published:2022-07-14 Issue:4 Volume:131 Page:217-223
ISSN:1742-7835
Container-title:Basic & Clinical Pharmacology & Toxicology
language:en
Short-container-title:Basic Clin Pharma Tox

Author:

Chen Xue¹²^ORCID,Yao Haidong²,Andrés Vicente³⁴,Bergo Martin O.²,Kashif Muhammad⁵

Affiliation:

1. Department of Plastic and Cosmetic Surgery, Tongji Hospital, Tongji Medical College Huazhong University of Science and Technology Wuhan China

2. Department of Biosciences and Nutrition Karolinska Institutet Huddinge Sweden

3. Centro Nacional de Investigaciones Cardiovasculares Carlos III (CNIC) Madrid Spain

4. CIBER de Enfermedades Cardiovasculares (CIBERCV) Madrid Spain

5. Center for Hematology and Regenerative Medicine, Department of Medicine, Huddinge Karolinska Institutet Huddinge Sweden

Funder

Medicinska Forskningsrådet

Fundació la Marató de TV3

European Regional Development Fund

Progeria Research Foundation

Publisher

Wiley

Subject

Pharmacology,Toxicology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/bcpt.13770

Reference36 articles.

1. Hutchinson–Gilford progeria syndrome: Review of the phenotype

2. Recurrent de novo point mutations in lamin A cause Hutchinson–Gilford progeria syndrome

3. Lamin A Truncation in Hutchinson-Gilford Progeria

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4. The role of prelamin A post-translational maturation in stress response and 53BP1 recruitment;Frontiers in Cell and Developmental Biology;2022-11-16

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